We assessed the effect of CAI in dextran sodiumsulfate-induced colitis.
2
The disruption of the epithelial barrier with dextran sodiumsulfate leads to increased IL-19 expression.
3
Separation was optimized by altering pH, the concentration of sodiumsulfate and the pairing ion.
4
Recent findings: Using the azoxymethane-dextran sodiumsulfate model, wound healing pathways seem to be required in the development of CAC.
5
Functional studies showed that defective IL-36R signalling causes high susceptibility to acute dextran sodiumsulfate colitis and impairs wound healing.